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Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 17
Published: 15 November 2023
10.55418/9781933477299-ch04
ISBN-10: 1-933477-29-6
ISBN: 978-1-933477-29-9
... nontransmural infarcts Acute transmural infarcts, following successful thrombolytic therapy Elevated catecholamine levels:  High-dose pressors  Stress cardiomyopathy  Acute nontransmural infarcts  Stress cardiomyopathy  Pheochromocytoma  Acute subarachnoid hemorrhage  Acute brain death  Septic shock  Cocaine...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 17
Published: 15 November 2023
10.55418/9781933477299-ch18
ISBN-10: 1-933477-29-6
ISBN: 978-1-933477-29-9
...Table 18-1 DIAGNOSTIC CRITERIA FOR STRESS CARDIOMYOPATHY a All four of the following features are required for the diagnosis:  Transient left ventricular systolic dysfunction  Absence of obstructive coronary artery disease or evidence of plaque rupture  New...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY: APICAL BALLOONING  Left ventriculography shows dilat...
Published: 15 November 2023
STRESS CARDIOMYOPATHY: APICAL BALLOONING Figure 18-1 STRESS CARDIOMYOPATHY: APICAL BALLOONING Left ventriculography shows dilatation of the left ventricular apex, so-called “apical ballooning,” somewhat characteristic of this condition. More about this image found in STRESS CARDIOMYOPATHY: APICAL BALLOONING Left ventriculography shows dilat...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  The prominent left ventricular dilatation is relativ...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-2 STRESS CARDIOMYOPATHY The prominent left ventricular dilatation is relatively diffuse, manifesting in this section at the mid-ventricular level and suggesting widespread ventricular involvement. More about this image found in STRESS CARDIOMYOPATHY The prominent left ventricular dilatation is relativ...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  Approximately half of the myocytes, distributed thro...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-3 STRESS CARDIOMYOPATHY Approximately half of the myocytes, distributed throughout, demonstrate myocyte necrosis and thinning, with indistinct contraction bands. This patient presented in heart failure after a home accident. More about this image found in STRESS CARDIOMYOPATHY Approximately half of the myocytes, distributed thro...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  The patient had an acute episode of heart failure a ...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-4 STRESS CARDIOMYOPATHY The patient had an acute episode of heart failure a year prior to transplant after an episode of severe emotional stress. The coronary arteries were normal. Low magnification (above) shows an apical aneurysm. High magnification (right) exhib... More about this image found in STRESS CARDIOMYOPATHY The patient had an acute episode of heart failure a ...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  There are confluent areas of necrosis with identifia...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-5 STRESS CARDIOMYOPATHY There are confluent areas of necrosis with identifiable contraction bands and a sparse inflammatory infiltrate. More about this image found in STRESS CARDIOMYOPATHY There are confluent areas of necrosis with identifia...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  Contraction bands and marked myocyte attenuation wit...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-6 STRESS CARDIOMYOPATHY Contraction bands and marked myocyte attenuation with loss of cross striations. There is no inflammation. More about this image found in STRESS CARDIOMYOPATHY Contraction bands and marked myocyte attenuation wit...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY: “BRAIN DEATH” LESIONS  There are small groups of myo...
Published: 15 November 2023
STRESS CARDIOMYOPATHY: “BRAIN DEATH” LESIONS Figure 18-7 STRESS CARDIOMYOPATHY: “BRAIN DEATH” LESIONS There are small groups of myocytes (arrows) with flocculent necrotic cytoplasm surrounded by scant mononuclear inflammatory cells. More about this image found in STRESS CARDIOMYOPATHY: “BRAIN DEATH” LESIONS There are small groups of myo...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  Foci of myocyte loss, with minimal inflammation and ...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-8 STRESS CARDIOMYOPATHY Foci of myocyte loss, with minimal inflammation and myocyte necrosis, are seen in a patient who developed left ventricular dysfunction following chemotherapy. The focus in the middle of the field is a typical “brain death” lesion. More about this image found in STRESS CARDIOMYOPATHY Foci of myocyte loss, with minimal inflammation and ...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY  Top: Contraction band necrosis is seen without infla...
Published: 15 November 2023
STRESS CARDIOMYOPATHY Figure 18-9 STRESS CARDIOMYOPATHY Top: Contraction band necrosis is seen without inflammation. Cross striations are lost. Bottom: Complement C4d staining by immunohistochemistry highlights necrotic myocytes. More about this image found in STRESS CARDIOMYOPATHY Top: Contraction band necrosis is seen without infla...
Images
<span class="search-highlight">STRESS</span> CARDIOMYOPATHY: MYOCYTE CALCIFICATION  The necrotic cells are identi...
Published: 15 November 2023
STRESS CARDIOMYOPATHY: MYOCYTE CALCIFICATION Figure 18-10 STRESS CARDIOMYOPATHY: MYOCYTE CALCIFICATION The necrotic cells are identified by diffuse cytoplasmic calcification caused by renal failure and hypercalcemia. More about this image found in STRESS CARDIOMYOPATHY: MYOCYTE CALCIFICATION The necrotic cells are identi...
Images
DILATED CARDIOMYOPATHY: SARCOPLASMIC VACUOLIZATION AND LIPOFUSINOSIS  The m...
Published: 15 November 2023
DILATED CARDIOMYOPATHY: SARCOPLASMIC VACUOLIZATION AND LIPOFUSINOSIS Figure 14-13 DILATED CARDIOMYOPATHY: SARCOPLASMIC VACUOLIZATION AND LIPOFUSINOSIS The myocytes exhibit increased glycogen content as well as lipofuscin pigment, both indicators of chronic myocyte stress. More about this image found in DILATED CARDIOMYOPATHY: SARCOPLASMIC VACUOLIZATION AND LIPOFUSINOSIS The m...
Images
SEMILUNAR VALVES  A,B: Aortic valve, in simulated opened (A) and closed (B)...
Published: 15 November 2023
SEMILUNAR VALVES Figure 1-22 SEMILUNAR VALVES A,B: Aortic valve, in simulated opened (A) and closed (B) positions, illustrating the extra length of free edge needed to completely close the valve orifice centrally. C: Pulmonary valve, with thin, arch-like, fibrous ridges that provide strength alo... More about this image found in SEMILUNAR VALVES A,B: Aortic valve, in simulated opened (A) and closed (B)...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 18
Published: 01 January 2024
10.55418/9781933477367-07
ISBN-10: 1-933477-36-9
ISBN: 978-1-933477-36-7
... . 38. Haque   W , Verma   V , Suzanne Klimberg   V , et al . Clinical presentation, national practice patterns, and outcomes of breast adenomyoepithelioma . Breast J   2020 ; 26 : 653 - 60 . 39. Hayes   MM . Adenomyoepithelioma of the breast: a review stressing its propensity...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 18
Published: 01 January 2024
10.55418/9781933477367-08
ISBN-10: 1-933477-36-9
ISBN: 978-1-933477-36-7
... of cell division (tumor suppressor) Usually IBC, NST Apocrine differentiation reported Nonmedullary thyroid Endometrial Renal cell carcinoma Melanoma Colon Ataxia-telangiectasia ATM DNA damage response (DNA repair, apoptosis, cell cycle, stress response) No specific features noted...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 17
Published: 15 November 2023
10.55418/9781933477299-ch13
ISBN-10: 1-933477-29-6
ISBN: 978-1-933477-29-9
... failure with reduced ejection fraction (HFrEF). This may represent the end stage of dilated, stress-related and noncompaction forms of cardiomyopathy. Other types of cardiomyopathy (e.g., hypertrophic cardiomyopathy), particularly with progression of the disease, frequently culminate in systolic...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 17
Published: 15 November 2023
10.55418/9781933477299-ch05
ISBN-10: 1-933477-29-6
ISBN: 978-1-933477-29-9
... of intimal SMC at branch points, in areas of decreased shear stress Normal finding from late childhood Intimal xanthoma (fatty streak) Foamy macrophage accumulation AHA type I (not visible) AHA type II (grossly visible) Potentially Obstructive Lesions Smooth muscle cell-rich plaque SMC...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 17
Published: 15 November 2023
10.55418/9781933477299-ch23
ISBN-10: 1-933477-29-6
ISBN: 978-1-933477-29-9
.... As with other toxicities, oxidative stress and calcium homeostasis are probable mechanisms ( 38 , 39 ). As with the presentation and imaging findings, the pathology of alcohol-associated cardiotoxicity is largely indistinguishable from that observed in DCM ( fig. 23-12 ). Four-chamber dilatation is observed...
Book Chapter
Series: Atlases of Tumor and Non-Tumor Pathology, Series 5
Volume: 17
Published: 15 November 2023
10.55418/9781933477299-ch09
ISBN-10: 1-933477-29-6
ISBN: 978-1-933477-29-9
... of the prosthesis. Calcification begins within 8 years of implantation and greatly increases after 10 years. Risk factors include young recipient age, glutaraldehyde fixation of the tissue valve, and mechanical stress. Pannus (or obstructive fibrous ingrowth) occurs on both mechanical...